ERITRODERMA
DEFINITION
Skin disorder characterized the Erythema in almost the entire body, usually accompanied by skuama.
SYNONYM
Eksfoliativa Dermatitis
PATHOPHYSIOLOGY
• Depending on the cause.
• At Eritroderma an increase in epidermal turn over, so the form skuama redundant.
• Skuama mainly contain protein, nucleic acid and free amino acids.
CLINICAL SYMPTOMS
• Occurs in a short time
• Fast expand
• Fever, chills, malaise
• The skin looks shiny, eritematus, Peel, palpable heat, itch, rough and thickened.
PATHOPHYSIOLOGY
Erythema: widening blood vessels → blood flow to skin
increased heat loss increases: →
» Cold/chills Sufferers
» Hipotermi (due to an increase in skin perfusion)
» Dehydration (due to increased evaporation of fluid)
» Temperature Setting.
Skuama loss can cause loss of protein (hipoproteinemia) and oedema.
CLINICAL SYMPTOMS
• Allergy Medication systemically
- Anamnesis: a history of medication/herbal medicine.
- Allergies arising in acute (about 10 days).
- A universal Erythema without skuama,
- After recovering recently arising erythema.
• Expansion of skin diseases
Psoriasis:
- Due to the disease itself or pengobtan is too strong.
- Erythema is uneven.
(Erythema has a predilection psoriasis)
Seboroika Dermatitis in infants (Leiner's disease)
- Dermatitis seboroika which extends
- Clearly unknown Etiology
- Age: > > > 4 – 20 weeks.
- KU patient well.
- Erythema universal accompanied skuama rough.
• Systemic diseases including malignancies
- Classified in the CTCL (Cutaneous T-Cell Lymphoma)
- > > > on seniors.
- Erythema universal meah smoldering + skuama and is very itchy.
- At 1/3 the sufferer: Lymphadenopathy, splenomegaly
superficial, alopecia, hyperpigmentation, hiperkeratotik
distrofia palmoplantar, nails.
- Laboratory: sezary cells called atipik Lymphoma
DIAGNOSIS
Clinical keradangan eritematus skin found along deskuamasi to ≥ 90% of the surface area of the body.
DIAGNOSIS OF APPEAL
Dermatitis,
Psoriasis vulgaris, Drug Eruption, lymphoma/Leukemia, Pityriasis Rubra
Pilaris, Pemfigus, Likhen Planus, Dermatofitosis, Skabies
THAT MAKES IT DIFFICULT
• Hipotermi
• Decompensation kordis
• Failure of peripheral circulation
• Tromboflebitis
• Secondary infections of the skin and the lungs
MANAGEMENT
• Fix the fluid body
• Elimination of precipitating factors
• Systemic Corticosteroids "tappering off"
• Antibiotics for secondary infections mencegaf
• Antihistamine/antipruritus
• Topical: coccos (oleum emolient, hydrocortisone 1%)
• High protein Diet
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